Skip to main content

Study of articles: Importance of diet in the pre and post-operative period in oral and maxillofacial surgeries

Study of articles: Importance of diet in the pre and post-operative period in oral and maxillofacial surgeries   In major and minor oral surgeries , there are three stages: preoperative, intraoperative, and postoperative (before, during, and after the procedure, respectively). Often, the most significant complications occur during the postoperative phase , including localized and diffuse pain, swelling, bruising, hyper and hypoglycemic episodes, lowered immunity, and, in extreme cases of neglect, patients may suffer from malnutrition , and, later on, fatal consequences. With this in mind, the quicker and less painful the healing process of intentional traumas caused by oral and maxillofacial surgeries, the better the patient's recovery. A suitable, nutritious diet with proper professional guidance can reduce healing time and contribute to postoperative success. This is possible because of cellular repair processes, which work better with a source of proteins and amino acids, the...

Everything you need to know about Clinical Periodontology - The Basics!

 Clinical Periodontology:

GENERAL PRINCIPLES:

4 phases:

1 - Assessment and diagnosis

Periogram, prophylaxis, plaque detection, diagnosis and oral hygiene instructions

  • 2 - Cause-related therapy
  • Scaling and root planing, non-surgical treatments (oral hygiene instructions) 3 - Reevaluation
  • 2 scenarios:
    • If the patient has improved, proceed to the 4th phase
    • If the patient has not improved, either return to phase 2 or consider surgical treatment if necessary. 4 - Periodontal support therapy
  • Periodic recalls every 1 year or, for more complex cases, every 6 months.

1

Periogram:

Complete mouth probing to identify bleeding points, probing depth (PD), gingival level (GL), calculate clinical attachment level (CAL), plaque index. Probing: North Carolina millimeter probe = Each mark represents 1mm, the first black mark indicates between 4 and 5mm, the second between 9 and 10mm, and the third between 14 and 15mm. Nabers probe: Used when assessing furcation involvement. This curved probe has two dark areas, with the first marking from 3 to 6mm and the second from 9 to 12mm. The North Carolina probe is used to measure both PD and GL. When inserted into the sulcus/gum pocket of the patient, it marks the PD, and the level of recession or swelling is the GL. The GL is calculated as follows: Recession is +mm, and swelling is -mm.

  • For example: Recession (GL) of 2mm and pocket depth (PD) of 4mm = 4 + 2 = 6 (CAL)
  • If there is gingival swelling (GL) of, for example, 2mm, it becomes: 4 (PD) - 2 (GL) = 2 (CAL)

If CAL is equal to or less than 3mm, it's a sulcus; if greater than 3mm, it's considered a periodontal pocket. Bleeding Index:

  • To understand this, we need to know the probed sites: Each tooth should be probed at a minimum of 6 sites (3 on the palatal and 3 on the buccal side).

The formula to calculate the bleeding index is as follows: Number of bleeding sites divided by probed sites times 100. If the index is less than or equal to 10%, it indicates health.

Bleeding Index=(Number of Probed SitesNumber of Bleeding Sites)×100

Plaque Index:

  • To calculate, we need to visualize the plaque.
  • Then, use the same formula, but instead of the number of bleeding sites, use the number of surfaces with plaque.
  • Dental Plaque Index=(Total Number of Surfaces EvaluatedNumber of Surfaces with Plaque)×100

Diagnosis:

Gingivitis:

  • A condition characterized by gingival inflammation due to biofilm (etiology).
  • More than 10% bleeding.
  • Can be localized or generalized; if up to 30% of teeth are involved, it's considered localized, and if more than 30%, it's generalized.

Non-biofilm-induced gingival diseases:

  • Hereditary/Medicamentous gingival fibromatosis
  • Burns
  • Systemic diseases
  • Fungal diseases: Candidiasis, Histoplasmosis, Aspergillosis
  • Viral diseases: Herpes, HPV, Varicella, Hand-foot-mouth disease
  • Bacterial diseases: Tuberculosis, Gonorrhea
  • Immune-inflammatory conditions
  • Neoplasms
  • Scurvy
  • Reactive processes
  • Traumatic injuries

Periodontitis:

  • A chronic inflammatory disease, multifactorial, associated with dysbiotic biofilm and characterized by loss of periodontal bone attachment.

CLASSIFICATION: Stages I, II, III, IV, and Grades A, B, C

  • Start by assessing clinical attachment loss.
  • Followed by radiographic bone loss.
  • If complex conditions like furcation lesions and advanced mobility are present, the stage is upgraded, always based on the worst scenario found.
  • Staging indicates disease severity.

Stages I, II, III, IV: Loss of less than 15% attachment, 1-2 mm = Stage I

Loss of 15-33% attachment, 3-4 mm = Stage II

Loss of more than 33% attachment (up to the middle third of the root), more than 5 mm with tooth loss of less than 4 teeth (due to periodontitis), possible furcation involvement grade 2 or 3 = Stage III

Bone loss beyond the midpoint of the root, tooth loss of 5 or more teeth due to periodontitis, severe ridge defects, generalized mobility grade 2 or 3, less than 20 remaining teeth = Stage IV

Grades A, B, C

  • Grade A: Determining factor: Direct - No progression of attachment loss in the last 5 years; if no evidence from 5 years ago, assess indirectly - Note the percentage of bone loss and divide it by the patient's age; if the result is less than or equal to 0.25mm, it's grade A. High biofilm accumulation and minimal periodontal destruction. Non-smoker and non-diabetic.


  • Grade B: Direct - Progression less than 2mm in 5 years Indirect - If bone loss calculation is between 0.25mm and 1mm = grade B Moderate biofilm accumulation and periodontal destruction, both moderate. Smokes less than 10 cigarettes per day and HbA1c <7% (glycated hemoglobin); not diabetic.


  • Grade C: Direct - Progression equal or greater than 2mm Indirect - If the bone loss calculation per age is more than 1mm Low biofilm for significant periodontal destruction. Smokes more than 10 cigarettes per day and HbA1c >7% (Diabetic).

Comments

Popular posts from this blog

Do you know what a furcation lesion is?! Come and see!

  Furcation Lesion: Furcation is the root area where there is a separation between two or more roots. A highly sensitive region to perforate, as it can potentially split the tooth in half. Furcation lesions are situations where there is a horizontal bone loss between the roots, due to periodontitis or endodontic problems, root fractures, and occlusal trauma. We classify furcation lesions into 3 classes: I, II, III . When the furcation lesion is class I, it means that the Nabers probe has penetrated up to 3mm, which is less than half of the tooth. If it's a class II furcation lesion, the Nabers probe reaches the middle of the furcation or goes beyond it, measuring between 6 and 9mm. Lastly. If it's a class III case, the probe passes to the other side of the tooth. Why is this detrimental to the patient's periodontal health? Because this is a location where a lot of food debris will accumulate, serving as a future food source for bacteria, and the gingival infectious...

Get to know a bit about guided tissue regeneration! It's magical!

  Guided Bone Regeneration: The process of guided bone regeneration involves the use of bone fragments and a membrane, both biocompatible. The most important materials are: Bone Grafts: These grafts are used to fill bone defects and expedite the bone regeneration process. There are four types of bone grafts: - Autogenous: These are bone fragments from the patient's own body. - Xenogeneic: These are bone fragments sourced from cattle. - Allogeneic: These are bone fragments from other human donors, typically obtained from bone banks. - Alloplastic: These are bone fragments from laboratories, synthetic grafts, and biocompatible materials. Membranes: The purpose of membranes is to establish a physical-mechanical barrier between the gingival epithelium and the root surface. This is crucial because direct interaction between these structures can prevent periodontal ligament cells from returning to their proper location. Instead of bone regeneration, it can lead to continued gingiva...